Ticagrelor

1. Drug Name

  • Generic Name: Ticagrelor

  • Brand Names: Brilinta, Brilique

2. Drug Classification

  • Class: Antiplatelet Agent

  • Subclass: P2Y12 Receptor Antagonist (Reversible)

3. Mechanism of Action

Ticagrelor is a direct-acting and reversible P2Y12 receptor antagonist, which inhibits adenosine diphosphate (ADP)-mediated activation of the P2Y12 receptor on platelets. By blocking this receptor, ticagrelor prevents ADP-induced platelet aggregation and thrombus formation. Unlike other P2Y12 inhibitors such as clopidogrel, ticagrelor does not require metabolic activation, allowing for faster onset and greater platelet inhibition.

4. Pharmacokinetics

  • Absorption: Ticagrelor has a bioavailability of approximately 36%. It reaches peak plasma concentrations within 1.5-3 hours after oral administration.

  • Distribution: The volume of distribution (Vd) is approximately 88 liters. Ticagrelor is highly protein-bound (99%).

  • Metabolism: Ticagrelor is metabolized primarily by the CYP3A4 enzyme in the liver to its active metabolite, which also inhibits the P2Y12 receptor.

  • Excretion: The elimination half-life is 6-12 hours for ticagrelor and around 8-13 hours for its active metabolite. Excretion occurs mainly through the biliary and hepatic routes.

  • Special Considerations:

    • Renal Impairment: No dose adjustment is needed for renal impairment, but close monitoring is advised.

    • Hepatic Impairment: Not recommended in severe hepatic impairment, and caution is advised in mild-to-moderate hepatic impairment.

5. Indications

  • Primary Indications:

    • Acute Coronary Syndrome (ACS): For the reduction of thrombotic cardiovascular events in patients with ACS, including those managed medically or with percutaneous coronary intervention (PCI).

    • Secondary Prevention of Cardiovascular Events: For patients with a history of myocardial infarction to reduce the risk of further cardiovascular events.

  • Specific Populations: Beneficial in patients with a history of ACS, especially those who may require rapid and reversible platelet inhibition.

6. Dosage and Administration

  • General Adult Dosing:

    • Acute Coronary Syndrome: Initial loading dose of 180 mg orally, followed by a maintenance dose of 90 mg twice daily.

    • Post-MI Secondary Prevention: After the first year following an MI, a dose of 60 mg twice daily may be used for long-term prevention.

  • Administration: Ticagrelor can be taken with or without food. Avoid concomitant use with high doses of aspirin (>100 mg daily) as it can reduce ticagrelor’s efficacy.

7. Contraindications

  • Absolute Contraindications:

    • History of intracranial hemorrhage.

    • Active pathological bleeding (e.g., peptic ulcer or severe GI bleeding).

    • Severe hepatic impairment.

  • Relative Contraindications:

    • Co-administration with strong CYP3A4 inhibitors or inducers.

    • High risk of bleeding or undergoing major surgery.

8. Warnings and Precautions

  • Bleeding Risk: Ticagrelor increases bleeding risk, especially in patients with recent surgery or a history of bleeding. Monitor for signs of bleeding and avoid use in patients with active bleeding conditions.

  • Dyspnea: Some patients may experience dyspnea as an adverse effect, which is usually mild and transient but may require clinical evaluation.

  • Bradyarrhythmias: Caution in patients with a history of bradycardia or conduction disturbances, as ticagrelor may cause transient pauses.

9. Adverse Effects

  • Common Adverse Effects (≥10%):

    • Dyspnea.

    • Bleeding, including minor bleeding such as epistaxis and contusions.

  • Less Common but Clinically Significant Side Effects:

    • Gastrointestinal bleeding, including peptic ulcers.

    • Bradycardia or heart block, especially in predisposed individuals.

  • Rare/Serious Adverse Reactions:

    • Intracranial hemorrhage, which can be life-threatening.

    • Significant or severe hypersensitivity reactions, including anaphylaxis.

10. Drug Interactions

  • Major Drug Interactions:

    • CYP3A4 Inhibitors (e.g., ketoconazole, ritonavir): These drugs increase ticagrelor plasma levels and bleeding risk. Avoid co-administration or consider alternatives.

    • CYP3A4 Inducers (e.g., rifampin, carbamazepine): These drugs decrease ticagrelor plasma levels, reducing efficacy.

    • Aspirin: Use caution with high doses of aspirin (>100 mg daily) as it may decrease ticagrelor effectiveness.

  • Food-Drug Interactions: None significant, although it can be taken with food to minimize gastrointestinal discomfort.

  • Interactions with Lab Tests: May affect platelet function tests and coagulation assays, though routine monitoring is not required.

11. Clinical Pharmacology

  • Pharmacodynamics: Ticagrelor achieves potent and rapid platelet inhibition through direct, reversible inhibition of the P2Y12 receptor. It has a higher efficacy than clopidogrel in reducing major adverse cardiovascular events but has an associated higher risk of bleeding.

12. Special Populations

  • Pregnancy: Ticagrelor is not recommended during pregnancy due to insufficient data on safety; bleeding risks are a concern for both mother and fetus.

  • Lactation: Not recommended during breastfeeding due to lack of safety data.

  • Geriatrics: No specific dose adjustments are required, but careful monitoring for bleeding is essential due to higher bleeding risks in older adults.

  • Pediatrics: Safety and efficacy have not been established in pediatric patients.

13. Therapeutic Uses

  • Primary Use: Ticagrelor is primarily used for acute management of ACS and secondary prevention in patients with a history of myocardial infarction.

  • Combinational Therapy: Often combined with aspirin (dual antiplatelet therapy) to optimize prevention of thrombotic events in high-risk cardiovascular patients.

14. Monitoring and Follow-Up

  • Bleeding Monitoring: Routine assessment for signs of bleeding, especially gastrointestinal and intracranial bleeding, is essential.

  • Liver Function Tests: Monitor liver enzymes, especially in patients with hepatic impairment.

  • Symptom Monitoring: Evaluate for dyspnea and bradyarrhythmic symptoms during therapy.

15. Overdose Management

  • Symptoms of Overdose: Overdose primarily results in excessive bleeding. Severe overdose may increase the risk of bleeding-related mortality.

  • Treatment Protocols:

    • Activated Charcoal: May be administered if the overdose is recent.

    • Supportive Measures: Includes fluid replacement, blood products, and platelet transfusions if indicated. Ticagrelor’s anticoagulant effect may persist for several days post-discontinuation due to its long half-life.

    • Reversal Agents: No specific antidote is available for ticagrelor; treatment focuses on supportive care and symptom management.

16. Patient Counseling Information

  • Key Points:

    • Inform patients to take ticagrelor exactly as prescribed and not to discontinue abruptly without consulting a healthcare provider, as it may increase cardiovascular risk.

    • Report any signs of bleeding, including unusual bruising, blood in stools or urine, or prolonged bleeding after cuts.

    • Notify all healthcare providers, including dentists, of ticagrelor use before any surgical or invasive procedure.

  • Seek Immediate Medical Attention: Patients should seek prompt care if they experience significant bleeding, severe shortness of breath, or symptoms suggesting an adverse reaction like unusual dizziness or palpitations.