Gemfibrozil

1. Drug Name

  • Generic Name: Gemfibrozil

  • Brand Names: Lopid

2. Drug Classification

  • Class: Fibrates

  • Subclass: Antilipemic agent

3. Mechanism of Action

  • Peroxisome Proliferator-Activated Receptor Alpha (PPAR-α) Agonist: Gemfibrozil primarily exerts its effects by activating PPAR-α, a nuclear receptor that regulates lipid metabolism. The activation of PPAR-α leads to the transcription of several genes involved in lipid metabolism, including those responsible for increasing lipoprotein lipase (LPL) activity and reducing the production of very low-density lipoproteins (VLDL).

    • Lipoprotein Lipase Activation: Gemfibrozil increases the activity of LPL, an enzyme responsible for the hydrolysis of triglycerides (TG) in chylomicrons and VLDL particles, leading to a reduction in circulating triglyceride levels.

    • Reduction of VLDL Production: By decreasing hepatic production of VLDL, the drug reduces overall triglyceride levels.

    • Increase in HDL Levels: Gemfibrozil also increases high-density lipoprotein (HDL) cholesterol levels by promoting the synthesis of apolipoprotein A1 (apoA1), which is a major component of HDL.

  • Lipid Modulation: The primary benefit of gemfibrozil is the reduction of triglycerides and the modest increase in HDL cholesterol, which is beneficial for patients with hypertriglyceridemia or mixed dyslipidemia.

4. Pharmacokinetics

  • Absorption:

    • Gemfibrozil is well absorbed from the gastrointestinal tract, with peak plasma concentrations reached within 1-2 hours after oral administration.

    • Its bioavailability is about 60%, and food intake can affect its absorption, slightly increasing plasma levels.

  • Distribution:

    • It is extensively protein-bound (approximately 99%) to plasma proteins, predominantly albumin.

    • Gemfibrozil has a moderate volume of distribution (Vd) and is widely distributed in the body.

  • Metabolism:

    • Gemfibrozil is extensively metabolized in the liver via the cytochrome P450 3A4 enzyme system (CYP3A4), producing active metabolites that contribute to its lipid-lowering effects.

  • Excretion:

    • Half-life (t½): The elimination half-life of gemfibrozil is approximately 1-2 hours, with its active metabolites having a longer half-life of around 10-20 hours.

    • Excretion: The drug and its metabolites are primarily excreted in the urine, with a minor fraction eliminated in feces.

  • Special Considerations:

    • Renal Impairment: The drug should be used with caution in patients with renal dysfunction, as clearance may be reduced.

    • Hepatic Impairment: Caution is also advised in patients with liver disease, as gemfibrozil is primarily metabolized in the liver.

5. Indications

  • Primary Indications:

    • Hypertriglyceridemia: Gemfibrozil is indicated for the treatment of hypertriglyceridemia (serum triglycerides ≥500 mg/dL) to reduce the risk of pancreatitis.

    • Mixed Dyslipidemia: It is used for treating mixed dyslipidemia, where there is both elevated triglycerides and low HDL cholesterol.

  • Off-label Uses:

    • Hypercholesterolemia: Gemfibrozil may be used in some patients with high cholesterol levels, but fibrates are generally not the first-line agents for this indication.

    • Cardiovascular Risk: It is occasionally used to reduce the risk of cardiovascular events in patients with a high level of triglycerides, though this use is not widely supported by major clinical guidelines.

6. Dosage and Administration

  • Adult Dosing:

    • Hypertriglyceridemia: The typical adult dose is 600 mg taken twice daily, 30 minutes before meals.

    • Mixed Dyslipidemia: Same dosing regimen as for hypertriglyceridemia.

  • Pediatric Dosing:

    • The safety and efficacy of gemfibrozil in children have not been well established, and its use in pediatric patients is generally not recommended.

  • Renal Adjustments:

    • Dose adjustments may be needed for patients with moderate renal impairment (creatinine clearance <50 mL/min). The drug is contraindicated in severe renal impairment.

  • Hepatic Adjustments:

    • Gemfibrozil should be used with caution in patients with hepatic dysfunction, as the liver is the primary site of metabolism.

7. Contraindications

  • Absolute Contraindications:

    • Severe Renal Impairment: Gemfibrozil is contraindicated in patients with severe renal impairment (creatinine clearance <30 mL/min).

    • Active Liver Disease: Contraindicated in patients with active liver disease, including cirrhosis or unexplained persistent liver enzyme elevations.

    • Gallbladder Disease: Gemfibrozil should not be used in patients with a history of gallstones or gallbladder disease.

  • Relative Contraindications:

    • Hypersensitivity: Contraindicated in patients with known hypersensitivity to gemfibrozil or any of its components.

    • Pregnancy: Category C for pregnancy; it should only be used if the potential benefit justifies the potential risk to the fetus.

    • Breastfeeding: It is not recommended during breastfeeding due to potential risks to the infant.

8. Warnings and Precautions

  • Rhabdomyolysis: There is an increased risk of myopathy and rhabdomyolysis, particularly when gemfibrozil is combined with statins. Myopathy symptoms such as muscle pain, tenderness, or weakness should be reported immediately.

  • Liver Function Monitoring: Liver function tests should be conducted before initiating treatment and periodically thereafter to monitor for liver enzyme elevations.

  • Gallstones: Gemfibrozil can increase the risk of gallstone formation, and patients with a history of gallstones should be monitored.

  • Renal Impairment: In patients with mild to moderate renal impairment, dose adjustments may be necessary.

  • Hypersensitivity Reactions: Rare allergic reactions such as rash, urticaria, or anaphylaxis have been reported.

9. Adverse Effects

  • Common Adverse Effects:

    • Gastrointestinal: Dyspepsia, nausea, abdominal pain, diarrhea, and constipation.

  • Less Common but Clinically Significant:

    • Liver Enzyme Elevations: Mild elevations in liver transaminases can occur, requiring periodic monitoring.

    • Myopathy: Muscle pain, weakness, and tenderness, which can progress to rhabdomyolysis, especially when used in combination with statins.

    • Fatigue: Some patients report general feelings of fatigue and malaise.

  • Rare/Serious Adverse Effects:

    • Rhabdomyolysis: Severe muscle breakdown that can lead to kidney damage, characterized by muscle pain, weakness, and dark-colored urine.

    • Pancreatitis: Though rare, gemfibrozil can cause pancreatitis, particularly in patients with predisposing factors.

10. Drug Interactions

  • Statins: The combination of gemfibrozil with statins significantly increases the risk of muscle toxicity (myopathy and rhabdomyolysis). The use of gemfibrozil with certain statins, such as simvastatin, is contraindicated.

  • Warfarin: Gemfibrozil may enhance the effects of warfarin, increasing the risk of bleeding. Close monitoring of prothrombin time (PT) and INR is recommended when used concomitantly.

  • Cyclosporine: The use of gemfibrozil with cyclosporine may increase cyclosporine levels, necessitating close monitoring and dose adjustments.

  • Oral Antidiabetic Agents: Caution should be exercised when gemfibrozil is used with oral hypoglycemic agents, as it may alter glucose metabolism.

  • Bile Acid Sequestrants: Gemfibrozil may reduce the absorption of bile acid sequestrants, and they should be taken at different times of the day.

11. Clinical Pharmacology

  • Pharmacodynamics: Gemfibrozil reduces triglycerides, increases HDL cholesterol, and lowers LDL cholesterol. It exerts its effects by increasing the activity of lipoprotein lipase and reducing hepatic VLDL production.

  • Additional Effects: Gemfibrozil also has some antioxidant and anti-inflammatory effects, which may contribute to its cardiovascular benefits.

12. Special Populations

  • Pregnancy: Category C; should only be used if the potential benefit justifies the potential risk.

  • Lactation: Contraindicated; gemfibrozil is excreted in breast milk.

  • Geriatrics: Older adults may have an increased risk of adverse effects, particularly myopathy and liver dysfunction.

  • Renal Impairment: Use with caution in renal impairment. Dose adjustments are necessary for moderate renal dysfunction.

  • Hepatic Impairment: Use with caution in hepatic dysfunction.

13. Therapeutic Uses

  • Primary Hypertriglyceridemia: Gemfibrozil is primarily used for reducing elevated triglycerides, especially when levels are over 500 mg/dL.

  • Mixed Dyslipidemia: Useful in patients with elevated triglycerides and low HDL cholesterol, typically in combination with other lipid-lowering therapies.

  • Cardiovascular Risk Reduction: Gemfibrozil may help reduce the risk of cardiovascular events, particularly in patients with high triglycerides.

14. Monitoring and Follow-Up

  • Liver Enzymes: Monitor for liver dysfunction before treatment and periodically during treatment.

  • Kidney Function: Monitor renal function, particularly in those with pre-existing renal disease.

  • Muscle Symptoms: Monitor for signs of muscle toxicity, particularly when used with statins.

15. Overdose Management

  • Symptoms of Overdose: Symptoms include gastrointestinal distress, liver dysfunction, and muscle pain.

  • Management: There is no specific antidote for gemfibrozil overdose. Treatment is symptomatic and supportive, focusing on monitoring liver and renal function.

16. Patient Counseling Information

  • Administration: Take gemfibrozil 30 minutes before meals.

  • Side Effects: Inform patients to report any unexplained muscle pain or tenderness, as it may indicate serious side effects such as rhabdomyolysis.

  • Liver Health: Advise patients to seek medical attention if they experience symptoms of liver problems, such as jaundice or dark urine.

  • Gallstone Risk: Gemfibrozil may increase the risk of gallstones, so notify the healthcare provider if new abdominal symptoms occur.